How cerebellar ataxia is treated
Treatment of cerebellar ataxia is not therapy for the impaired coordination of movement as such, but elimination of the primary pathology. That is why a treatment plan is drawn up only after the disease that caused the cerebellar ataxia has been identified.
Therefore, the treatment approach may include a variety of methods. These include:
- drug therapy: antibacterial, antiviral, and vasodilating drugs, angioprotectors, thrombolytics, antiplatelet agents, diuretics, B vitamins, adenosine triphosphate, piracetam, ginkgo biloba preparations, etc.;
- infusion therapy;
- hemosorption;
- surgical treatment;
- chemotherapy;
- radiation therapy, etc.
To reduce the severity of cerebellar ataxia, that is, to improve metabolism in the skeletal muscles and increase muscle tone and strength, a course of medical massage and physical rehabilitation is usually prescribed.
Physical rehabilitation makes it possible to adapt activity and facilitate motor learning. A physical rehabilitation program may include exercises aimed at improving:
- static balance;
- dynamic balance;
- coordination of the trunk and limbs.
Physical rehabilitation also helps prevent contractures.
At first, the patient performs the exercises only under a doctor's supervision, but as progress is made, gradually moves on to daily exercise at home. Regular exercise helps improve the patient's physical condition, but for safety the patient is advised to use a cane or walker, which reduce the likelihood of falls caused by impaired balance or poor coordination. Work is also done to increase muscle strength and overall endurance.
In rehabilitation, in addition to exercises, physiotherapy is used to reduce the severity of cerebellar ataxia. Transcranial micropolarization (TCMP) is one of the methods used to strengthen muscles and improve coordination of movements; it helps maintain physical fitness, improve coordination and balance, and prevent complications.
Treatment of impaired coordination of movements in cerebellar damage in elderly people may reduce the severity of ataxia but will not eliminate it completely. Also, the result of therapy in patients with a single focal lesion is much more noticeable than in patients with degenerative diseases of the central nervous system.